Is Low Fat & High Fructose Causing Metabolic Syndrome?

Via Twitter, Beth from Weight Maven blog asked me if I read That Paleo Guy and a recent paper he blogged on.  The answers would be no and no, but I have now ;-)    The title of the study is:  Is the metabolic syndrome caused by a high fructose, and relatively low fat, low cholesterol diet?

 
The following stuck out at me from the conclusion of the paper:

In conclusion, we would urge medical practitioners to encourage individuals exhibiting MetS to strongly limit the consumption of dietary fructose [75] and other high-glycemic-index carbohydrates, and to stop discouraging them from consuming foods rich in cholesterol [76].

Now I don't know what medical practitioners are out there pushing fructose on people, but the recommendations to consume fruit are a far far cry from endorsing the consumption of sugar sweetened beverages and lots of juice.   I do think it's a crime that fruit juices are considered the equivalent of a piece of fruit to many nutritionists, but that aside, it is darned near impossible to eat 50g of fructose per day eating just whole fruits, let alone the 100g and upwards amounts in liquid form often used in studies.  There have also been scant few isocaloric studies done with fructose. 

In any case, as I read the study, I couldn't help getting "flashbacks" of sorts of the "Cafeteria Rat" study I blogged about over a year ago.  There is so much to that study that, yes, was in rats, but so are a heckuvalot of studies on MetSyn.   I'll be bumping the three posts directly referencing the study, and in a few days I'll publish the rest of this post that is in the hopper.  Here are the links anyway:

http://carbsanity.blogspot.com/2011/02/why-we-get-fat-lessons-from-cafeteria.html 

http://carbsanity.blogspot.com/2011/03/why-we-get-fat-lessons-from-obese.html

http://carbsanity.blogspot.com/2011/03/why-we-get-sick-fat-lessons-from.html

The last one deals with the effects that were seen on the liver, fat tissue and pancreatic cells and is probably most pertinent to a discussion of the fructose/low cholesterol hypothesis.