Non-Alcoholic Fatty Liver Disease: NAFLD. Along with diabetes, a disease on the rise in the US and around the globe where the obesity epidemic is rearing it's ugly head. Near as I can tell, Peter/Hyperlipid subscribes to a "damaged liver" hypothesis of obesity and diabetes. That the liver gets damaged, throws your insulin and glucose outta whack, and then you get fat. There can be no doubt that damage to the liver is an integral part of the metabolic syndrome(s) and the insulin resistance that underlies it. So the question is, do we take it seriously in all contexts? Or do we try to explain away inconvenient results when it is one's favored advocated diet that might possibly be a culprit?
Sadly, Peter seems bound and determined to spin ever more far-fetched mechanisms with which to explain away inconvenient results. Having nothing to do with fatty liver, Peter found himself unable to backtrack on his definitive statement that it was fasting insulin levels that determined fat loss. So bad was the evidence to counter this that he had to resort to manipulating study data to make his point. It seems where high fat rodent studies are concerned, Peter was hell bent on blaming transfatty acids. Now I'm no fan of the transfat, and they are indeed evil doers in any body, but they can't be blamed for everything. Indeed one just needs to look around to find high fat rodent diets that are not high in transfats. Enter the other bad guy by which we can dismiss inconvenient results ... veggie oil PUFA. True, many high fat rodent diets are very high PUFA ... but there are many that are based on lard. You know, the stuff us moist eyed female types should wallow in to enter the glories of Slimville via the Ketogenic Highway?!
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